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Author:  Timur, Zehra Kevser

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Issue DateTitleAuthor(s)
12020GM2 ganglioside accumulation causes neuroinflammation and behavioral alterations in a mouse model of early onset Tay-Sachs diseaseAkyıldız Demir, Seçil; Timur, Zehra Kevser ; Ateş, Nurselin ; Martinez, Luis Alarcon; Seyrantepe, Volkan 
2Dec-2017Immunohistochemical, biochemical and imaging-mass spectrometric analysis of brain tissues of mice with combined deficiencies of ß-hexosaminidase a, sialidase Neu4 and GM2-apTimur, Zehra Kevser 
32016Lysosomal Cathepsin A plays a significant role in the processing of endogenous bioactive peptidesTimur, Zehra Kevser ; Akyıldız Demir, Seçil; Seyrantepe, Volkan 
4Jan-2018Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs diseaseSeyrantepe, Volkan ; Akyıldız Demir, Seçil; Timur, Zehra Kevser ; Von Gerichten, Johanna; Marsching, Christian; Erdemli, Esra; Öztaş, Emin; Takahashi, Kohta; Yamaguchi, Kazunori; Ateş, Nurselin ; Dönmez Demir, Buket; Dalkara, Turgay; Erich, Katrin; Hopf, Carsten; Sandhoff, Roger; Miyagi, Taeko
5Aug-2015Neuraminidase-1 contributes significantly to the degradation of neuronal B-series gangliosides but not to the bypass of the catabolic block in Tay-Sachs mouse modelsTimur, Zehra Kevser ; Akyıldız Demir, Seçil; Marsching, C.; Sandhoff, R.; Seyrantepe, Volkan 
62021Sialidase neu4 deficiency is associated with neuroinflammation in miceTimur, Zehra Kevser ; İnci, Orhan Kerim; Akyıldız Demir, Seçil; Seyrantepe, Volkan